Cardiovascular dysautonomia in Parkinson's disease and multiple system atrophy[This article has been retracted]
Identifieur interne : 001345 ( Main/Exploration ); précédent : 001344; suivant : 001346Cardiovascular dysautonomia in Parkinson's disease and multiple system atrophy[This article has been retracted]
Auteurs : H. Oka [Japon] ; S. Mochio [Japon] ; M. Yoshioka [Japon] ; M. Morita [Japon] ; K. Onouchi [Japon] ; K. Inoue [Japon]Source :
- Acta Neurologica Scandinavica [ 0001-6314 ] ; 2006-04.
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Abstract
Objectives – To determine whether Parkinson's disease (PD) can be distinguished from multiple system atrophy (MSA) on the basis of the assessment of iodine‐123 meta‐iodobenzylguanidine (123I‐MIBG) radioactivity in heart and cardiovascular autonomic function. Patients and methods – Seventeen patients with MSA, 39 with PD, and 25 healthy volunteers underwent 123I‐MIBG scintigraphy and hemodynamic autonomic function tests using Valsalva maneuver (VM). Baroreceptor reflex sensitivity (BRS) was measured using the slope of the relation between RR interval and blood pressure during the fourth phase. Results – 123I‐MIBG radioactivity in heart of patients with PD was lower than that of control subjects and patients with MSA, but there was some overlap between PD and MSA. BRS in patients with PD who had a 123I‐MIBG radioactivity similar to that in MSA was larger than that in patients with MSA, with no overlap in any patient. Conclusion – Assessment of BRS may be useful for differentiating between MSA and PD that had a 123I‐MIBG radioactivity similar to MSA.
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DOI: 10.1111/j.1600-0404.2005.00526.x
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Oka</name><affiliation wicri:level="3"><country xml:lang="fr">Japon</country><wicri:regionArea>Department of Neurology, Aoto Hospital, Tokyo</wicri:regionArea><placeName><settlement type="city">Tokyo</settlement></placeName></affiliation></author><author><name sortKey="Mochio, S" sort="Mochio, S" uniqKey="Mochio S" first="S." last="Mochio">S. Mochio</name><affiliation wicri:level="3"><country xml:lang="fr">Japon</country><wicri:regionArea>Third Hospital, Tokyo</wicri:regionArea><placeName><settlement type="city">Tokyo</settlement></placeName></affiliation></author><author><name sortKey="Yoshioka, M" sort="Yoshioka, M" uniqKey="Yoshioka M" first="M." last="Yoshioka">M. Yoshioka</name><affiliation wicri:level="3"><country xml:lang="fr">Japon</country><wicri:regionArea>Main Hospital, Jikei University School of Medicine, Tokyo</wicri:regionArea><placeName><settlement type="city">Tokyo</settlement></placeName></affiliation></author><author><name sortKey="Morita, M" sort="Morita, M" uniqKey="Morita M" first="M." last="Morita">M. Morita</name><affiliation wicri:level="3"><country xml:lang="fr">Japon</country><wicri:regionArea>Department of Neurology, Aoto Hospital, Tokyo</wicri:regionArea><placeName><settlement type="city">Tokyo</settlement></placeName></affiliation></author><author><name sortKey="Onouchi, K" sort="Onouchi, K" uniqKey="Onouchi K" first="K." last="Onouchi">K. Onouchi</name><affiliation wicri:level="3"><country xml:lang="fr">Japon</country><wicri:regionArea>Department of Neurology, Aoto Hospital, Tokyo</wicri:regionArea><placeName><settlement type="city">Tokyo</settlement></placeName></affiliation></author><author><name sortKey="Inoue, K" sort="Inoue, K" uniqKey="Inoue K" first="K." last="Inoue">K. Inoue</name><affiliation wicri:level="3"><country xml:lang="fr">Japon</country><wicri:regionArea>Main Hospital, Jikei University School of Medicine, Tokyo</wicri:regionArea><placeName><settlement type="city">Tokyo</settlement></placeName></affiliation></author></analytic><monogr></monogr><series><title level="j">Acta Neurologica Scandinavica</title><idno type="ISSN">0001-6314</idno><idno type="eISSN">1600-0404</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="2006-04">2006-04</date><biblScope unit="volume">113</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="221">221</biblScope><biblScope unit="page" to="227">227</biblScope></imprint><idno type="ISSN">0001-6314</idno></series><idno type="istex">38A3ACF8E2C7B7061DBAA6475488709DF987C765</idno><idno type="DOI">10.1111/j.1600-0404.2005.00526.x</idno><idno type="ArticleID">ANE526</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0001-6314</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Parkinson's disease</term><term>Valsalva maneuver</term><term>baroreceptor reflex sensitivity</term><term>cardiac iodine‐123 meta‐iodobenzylguanidine (123I‐MIBG) scintigraphy</term><term>cardiovascular dysautonomia</term><term>multiple system atrophy</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Objectives – To determine whether Parkinson's disease (PD) can be distinguished from multiple system atrophy (MSA) on the basis of the assessment of iodine‐123 meta‐iodobenzylguanidine (123I‐MIBG) radioactivity in heart and cardiovascular autonomic function. Patients and methods – Seventeen patients with MSA, 39 with PD, and 25 healthy volunteers underwent 123I‐MIBG scintigraphy and hemodynamic autonomic function tests using Valsalva maneuver (VM). Baroreceptor reflex sensitivity (BRS) was measured using the slope of the relation between RR interval and blood pressure during the fourth phase. Results – 123I‐MIBG radioactivity in heart of patients with PD was lower than that of control subjects and patients with MSA, but there was some overlap between PD and MSA. BRS in patients with PD who had a 123I‐MIBG radioactivity similar to that in MSA was larger than that in patients with MSA, with no overlap in any patient. 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Yoshioka</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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